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Synchronous Systolic Subcellular Ca(2+)-Elevations Underlie Ventricular Arrhythmia in Drug-Induced Long QT Type 2
BACKGROUND: Repolarization-delay is a common clinical problem which can promote ventricular arrhythmias. In myocytes, abnormal sarcoplasmic reticulum Ca(2+)-release is proposed as the mechanism that causes early afterdepolarizations, the cellular equivalent of ectopic-activity in drug-induced long Q...
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| Publié dans: | Circ Arrhythm Electrophysiol |
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| Auteurs principaux: | , , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
2015
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4472565/ https://ncbi.nlm.nih.gov/pubmed/25722252 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCEP.114.002214 |
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