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Synchronous Systolic Subcellular Ca(2+)-Elevations Underlie Ventricular Arrhythmia in Drug-Induced Long QT Type 2

BACKGROUND: Repolarization-delay is a common clinical problem which can promote ventricular arrhythmias. In myocytes, abnormal sarcoplasmic reticulum Ca(2+)-release is proposed as the mechanism that causes early afterdepolarizations, the cellular equivalent of ectopic-activity in drug-induced long Q...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Circ Arrhythm Electrophysiol
Päätekijät: Kim, Jong J., Němec, Jan, Li, Qiao, Salama, Guy
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2015
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4472565/
https://ncbi.nlm.nih.gov/pubmed/25722252
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCEP.114.002214
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