טוען...
CMTX1 patients’ cells present genomic instability corrected by CamKII inhibitors
BACKGROUND: We previously described that fibroblasts from animal models of CMTX1 present genomic instability and poor connexon activity. In vivo, these transgenic mice present motor deficits. This phenotype could be significantly reverted by treatment with (CamKII) inhibitors. The objective of this...
שמור ב:
| הוצא לאור ב: | Orphanet J Rare Dis |
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| Main Authors: | , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
BioMed Central
2015
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4460704/ https://ncbi.nlm.nih.gov/pubmed/25947624 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13023-015-0270-5 |
| תגים: |
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