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CMTX1 patients’ cells present genomic instability corrected by CamKII inhibitors
BACKGROUND: We previously described that fibroblasts from animal models of CMTX1 present genomic instability and poor connexon activity. In vivo, these transgenic mice present motor deficits. This phenotype could be significantly reverted by treatment with (CamKII) inhibitors. The objective of this...
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| Publicado no: | Orphanet J Rare Dis |
|---|---|
| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4460704/ https://ncbi.nlm.nih.gov/pubmed/25947624 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13023-015-0270-5 |
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