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Neuronal Cx3cr1 Deficiency Protects against Amyloid β-Induced Neurotoxicity
Cx3cr1, the receptor for the chemokine Cx3cl1 (fractalkine), has been implicated in the progression and severity of Alzheimer’s disease-like pathology in mice, but the underlying mechanisms remain unclear. A complicating factor is that Cx3cr1 has been demonstrated in both neurons and microglia. Here...
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| Veröffentlicht in: | PLoS One |
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| Hauptverfasser: | , , , , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Public Library of Science
2015
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4454597/ https://ncbi.nlm.nih.gov/pubmed/26038823 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0127730 |
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