Mitochondrial dysfunction induced by a post-translationally modified amyloid linked to a familial mutation in an alternative model of neurodegeneration

Documenti analoghi

• Differential contribution of isoaspartate post-translational modifications to the fibrillization and toxic properties of amyloid β and the Asn(23) Iowa mutation
  di: Fossati, Silvia, et al.
  Pubblicazione: (2013)
• Oxidative stress and mitochondria-mediated cell death mechanisms triggered by the familial Danish dementia ADan amyloid
  di: Todd, Krysti, et al.
  Pubblicazione: (2015)
• Insights into Caspase-Mediated Apoptotic Pathways Induced by Amyloid-β in Cerebral Microvascular Endothelial Cells
  di: Fossati, Silvia, et al.
  Pubblicazione: (2012)
• Amyloidosis Associated with Cerebral Amyloid Angiopathy: Cell Signaling Pathways Elicited in Cerebral Endothelial Cells
  di: Ghiso, Jorge, et al.
  Pubblicazione: (2014)
• The carbonic anhydrase inhibitor methazolamide prevents amyloid beta-induced mitochondrial dysfunction and caspase activation protecting neuronal and glial cells in vitro and in the mouse brain
  di: Fossati, Silvia, et al.
  Pubblicazione: (2015)