Nrf2-Regulated Signaling Is Crucial for Alveolar Macrophage–Mediated Efferocytosis during Hyperoxic Lung Injury and Repair

Podobne zapisy

• PI3K-AKT Signaling via Nrf2 Protects against Hyperoxia-Induced Acute Lung Injury, but Promotes Inflammation Post-Injury Independent of Nrf2 in Mice
  od: Reddy, Narsa M., i wsp.
  Wydane: (2015)
• Alveolar macrophage procoagulant activity is increased in acute hyperoxic lung injury.
  od: Tipping, P. G., i wsp.
  Wydane: (1988)
• Preconditioning the immature lung with enhanced Nrf2 activity protects against oxidant-induced hypoalveolarization in mice
  od: Tamatam, Chandra M., i wsp.
  Wydane: (2020)
• The Lung Alveolar Lipofibroblast: An Evolutionary Strategy Against Neonatal Hyperoxic Lung Injury
  od: Rehan, Virender K., i wsp.
  Wydane: (2014)
• Conditional Deletion of Nrf2 in Airway Epithelium Exacerbates Acute Lung Injury and Impairs the Resolution of Inflammation
  od: Reddy, Narsa M., i wsp.
  Wydane: (2011)