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CD11b is protective in complement-mediated immune complex glomerulonephritis
In chronic serum sickness, glomerular immune complexes form, yet C57BL/6 mice do not develop glomerulonephritis unless complement factor H (CfH) is absent, indicating the relevance of complement regulation. Complement receptor 3 (CD11b) and Fcγ receptors on leukocytes, and CfH on platelets, can bind...
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| Publicado no: | Kidney Int |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4424815/ https://ncbi.nlm.nih.gov/pubmed/25565310 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ki.2014.373 |
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