CD11b is protective in complement-mediated immune complex glomerulonephritis

In chronic serum sickness, glomerular immune complexes form, yet C57BL/6 mice do not develop glomerulonephritis unless complement factor H (CfH) is absent, indicating the relevance of complement regulation. Complement receptor 3 (CD11b) and Fcγ receptors on leukocytes, and CfH on platelets, can bind...

Fuld beskrivelse

Na minha lista:
Bibliografiske detaljer
Udgivet i:Kidney Int
Main Authors: Alexander, Jessy J, Chaves, Lee D, Chang, Anthony, Jacob, Alexander, Ritchie, Maria, Quigg, Richard J
Format: Artigo
Sprog:Inglês
Udgivet: Nature Publishing Group 2015
Fag:
Basic Research
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4424815/
https://ncbi.nlm.nih.gov/pubmed/25565310
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ki.2014.373
Tags: Tilføj Tag
Ingen Tags, Vær først til at tagge denne postø!

Lignende værker