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Pain-enhancing mechanism through interaction between TRPV1 and anoctamin 1 in sensory neurons
The capsaicin receptor transient receptor potential cation channel vanilloid 1 (TRPV1) is activated by various noxious stimuli, and the stimuli are converted into electrical signals in primary sensory neurons. It is believed that cation influx through TRPV1 causes depolarization, leading to the acti...
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| Pubblicato in: | Proc Natl Acad Sci U S A |
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| Autori principali: | , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
National Academy of Sciences
2015
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4413337/ https://ncbi.nlm.nih.gov/pubmed/25848051 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1421507112 |
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