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Pain-enhancing mechanism through interaction between TRPV1 and anoctamin 1 in sensory neurons
The capsaicin receptor transient receptor potential cation channel vanilloid 1 (TRPV1) is activated by various noxious stimuli, and the stimuli are converted into electrical signals in primary sensory neurons. It is believed that cation influx through TRPV1 causes depolarization, leading to the acti...
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| Vydáno v: | Proc Natl Acad Sci U S A |
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| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4413337/ https://ncbi.nlm.nih.gov/pubmed/25848051 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1421507112 |
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