Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna—A Randomized, Cross-Over Clinical Study

Ítems similars

• Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna-A Randomized, Cross-Over Clinical Study.
  per: John Hayslip, et al.
  Publicat: (2015-01-01)
• Superoxide induces protein oxidation in plasma and TNF-α elevation in macrophage culture: Insights into mechanisms of neurotoxicity following doxorubicin chemotherapy
  per: Keeney, Jeriel T. R., et al.
  Publicat: (2015)
• Doxorubicin-induced elevated oxidative stress and neurochemical alterations in brain and cognitive decline: protection by MESNA and insights into mechanisms of chemotherapy-induced cognitive impairment (“chemobrain”)
  per: Keeney, Jeriel T. R., et al.
  Publicat: (2018)
• The Triangle of Death of Neurons: Oxidative Damage, Mitochondrial Dysfunction, and Loss of Choline-Containing Biomolecules in Brains of Mice Treated with Doxorubicin. Advanced Insights into Mechanisms of Chemotherapy Induced Cognitive Impairment (“Chemobrain”) Involving TNF-α
  per: REN, Xiaojia, et al.
  Publicat: (2018)
• Loss of Mrp1 Potentiates Doxorubicin-Induced Cytotoxicity in Neonatal Mouse Cardiomyocytes and Cardiac Fibroblasts
  per: Zhang, Wei, et al.
  Publicat: (2016)