Superoxide induces protein oxidation in plasma and TNF-α elevation in macrophage culture: Insights into mechanisms of neurotoxicity following doxorubicin chemotherapy

Míreanna Comhchosúla

• The Triangle of Death of Neurons: Oxidative Damage, Mitochondrial Dysfunction, and Loss of Choline-Containing Biomolecules in Brains of Mice Treated with Doxorubicin. Advanced Insights into Mechanisms of Chemotherapy Induced Cognitive Impairment (“Chemobrain”) Involving TNF-α
  le: REN, Xiaojia, et al.
  Foilsithe: (2018)
• Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna-A Randomized, Cross-Over Clinical Study.
  le: John Hayslip, et al.
  Foilsithe: (2015-01-01)
• Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna—A Randomized, Cross-Over Clinical Study
  le: Hayslip, John, et al.
  Foilsithe: (2015)
• Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna-A Randomized, Cross-Over Clinical Study.
  le: John Hayslip, et al.
  Foilsithe: (2015-01-01)
• Doxorubicin-induced elevated oxidative stress and neurochemical alterations in brain and cognitive decline: protection by MESNA and insights into mechanisms of chemotherapy-induced cognitive impairment (“chemobrain”)
  le: Keeney, Jeriel T. R., et al.
  Foilsithe: (2018)