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Metal-deficient SOD1 in amyotrophic lateral sclerosis
Mutations to the ubiquitous antioxidant enzyme Cu/Zn superoxide dismutase (SOD1) were the first established genetic cause of the fatal, adult-onset neurodegenerative disease amyotrophic lateral sclerosis (ALS). It is widely accepted that these mutations do not cause ALS via a loss of antioxidant fun...
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| Yayımlandı: | J Mol Med (Berl) |
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| Asıl Yazarlar: | , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Springer Berlin Heidelberg
2015
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4408375/ https://ncbi.nlm.nih.gov/pubmed/25754173 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00109-015-1273-3 |
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