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Golgi defects enhance APP amyloidogenic processing in Alzheimer’s disease
Increased amyloid beta (Aβ) production by sequential cleavage of the amyloid precursor protein (APP) by the β- and γ-secretases contributes to the etiological basis of Alzheimer’s disease (AD). This process requires APP and the secretases to be in the same subcellular compartments, such as the endos...
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| Publicado no: | Bioessays |
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| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4407201/ https://ncbi.nlm.nih.gov/pubmed/25546412 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/bies.201400116 |
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