The role of alterations in mitochondrial dynamics and PGC-1α over-expression in fast muscle atrophy following hindlimb unloading

The mechanisms triggering disuse muscle atrophy remain of debate. It is becoming evident that mitochondrial dysfunction may regulate pathways controlling muscle mass. We have recently shown that mitochondrial dysfunction plays a major role in disuse atrophy of soleus, a slow, oxidative muscle. Here...

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Vydáno v:J Physiol
Hlavní autoři: Cannavino, Jessica, Brocca, Lorenza, Sandri, Marco, Grassi, Bruno, Bottinelli, Roberto, Pellegrino, Maria Antonietta
Médium: Artigo
Jazyk:Inglês
Vydáno: BlackWell Publishing Ltd 2015
Témata:
Muscle
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4405755/
https://ncbi.nlm.nih.gov/pubmed/25565653
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2014.286740
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