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The role of alterations in mitochondrial dynamics and PGC-1α over-expression in fast muscle atrophy following hindlimb unloading

The mechanisms triggering disuse muscle atrophy remain of debate. It is becoming evident that mitochondrial dysfunction may regulate pathways controlling muscle mass. We have recently shown that mitochondrial dysfunction plays a major role in disuse atrophy of soleus, a slow, oxidative muscle. Here...

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Detalhes bibliográficos
Publicado no:J Physiol
Main Authors: Cannavino, Jessica, Brocca, Lorenza, Sandri, Marco, Grassi, Bruno, Bottinelli, Roberto, Pellegrino, Maria Antonietta
Formato: Artigo
Idioma:Inglês
Publicado em: BlackWell Publishing Ltd 2015
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4405755/
https://ncbi.nlm.nih.gov/pubmed/25565653
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2014.286740
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