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ACEI attenuates the progression of CCl(4)-induced rat hepatic fibrogenesis by inhibiting TGF-β1, PDGF-BB, NF-κB and MMP-2,9
AIM: Angiotensin II has pro-fibrotic function in the liver. Blockade of the renin-angiotensin-aldosterone-system (RAAS) attenuates hepatic fibrosis. The aim of the present study was to determine the mechanism of angiotensin-converting enzyme inhibitor (ACEI) on the progression of rat hepatic fibrosi...
Uloženo v:
| Vydáno v: | World J Gastroenterol |
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| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Baishideng Publishing Group Inc
2005
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4398726/ https://ncbi.nlm.nih.gov/pubmed/16097048 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3748/wjg.v11.i31.4807 |
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