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Identification of p18(INK4c) as a Tumor Suppressor Gene in Glioblastoma Multiforme

Genomic alterations leading to aberrant activation of cyclin/cyclin-dependent kinase (cdk) complexes drive the pathogenesis of many common human tumor types. In the case of glioblastoma multiforme (GBM), these alterations are most commonly due to homozygous deletion of p16(INK4a) and less commonly d...

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Bibliografische gegevens
Gepubliceerd in:Cancer Res
Hoofdauteurs: Solomon, David A., Kim, Jung-Sik, Jenkins, Sultan, Ressom, Habtom, Huang, Michael, Coppa, Nicholas, Mabanta, Lauren, Bigner, Darell, Yan, Hai, Jean, Walter, Waldman, Todd
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2008
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4394609/
https://ncbi.nlm.nih.gov/pubmed/18381405
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-07-6388
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