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Perinatal Nicotine Exposure Suppresses PPARγ Epigenetically in Lung Alveolar Interstitial Fibroblasts

Due to the active inhibition of the adipogenic programming, the default destiny of the developing lung mesenchyme is to acquire a myogenic phenotype. We have previously shown that perinatal nicotine exposure, by down-regulating PPARγ expression, accentuates this property, culminating in myogenic pul...

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Publicado en:Mol Genet Metab
Autores principales: Gong, M., Liu, J., Sakurai, R., Corre, A., Anthony, S., Rehan, V. K.
Formato: Artigo
Lenguaje:Inglês
Publicado: 2015
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4390504/
https://ncbi.nlm.nih.gov/pubmed/25661292
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ymgme.2015.01.004
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