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Perinatal Nicotine Exposure Suppresses PPARγ Epigenetically in Lung Alveolar Interstitial Fibroblasts
Due to the active inhibition of the adipogenic programming, the default destiny of the developing lung mesenchyme is to acquire a myogenic phenotype. We have previously shown that perinatal nicotine exposure, by down-regulating PPARγ expression, accentuates this property, culminating in myogenic pul...
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| Udgivet i: | Mol Genet Metab |
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| Main Authors: | , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2015
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4390504/ https://ncbi.nlm.nih.gov/pubmed/25661292 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ymgme.2015.01.004 |
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