Advanced glycation end products contribute to amyloidosis in Alzheimer disease.

Alzheimer disease (AD) is characterized by deposits of an aggregated 42-amino-acid beta-amyloid peptide (beta AP) in the brain and cerebrovasculature. After a concentration-dependent lag period during in vitro incubations, soluble preparations of synthetic beta AP slowly form fibrillar aggregates th...

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Detalhes bibliográficos
Main Authors: Vitek, M P, Bhattacharya, K, Glendening, J M, Stopa, E, Vlassara, H, Bucala, R, Manogue, K, Cerami, A
Formato: Artigo
Idioma:Inglês
Publicado em: 1994
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC43869/
https://ncbi.nlm.nih.gov/pubmed/8197133
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