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Mechanisms of angiotensin II stimulation of NCC are time-dependent in mDCT15 cells
Angiotensin II (ANG II) increases thiazide-sensitive sodium-chloride cotransporter (NCC) activity both acutely and chronically. ANG II has been implicated as a switch that turns WNK4 from an inhibitor of NCC into an activator of NCC, and ANG II's effect on NCC appears to require WNK4. Chronical...
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| Vydáno v: | Am J Physiol Renal Physiol |
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| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Physiological Society
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4385889/ https://ncbi.nlm.nih.gov/pubmed/25651566 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00465.2014 |
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