The intellectual disability protein RAB39B selectively regulates GluA2 trafficking to determine synaptic AMPAR composition

RAB39B is a member of the RAB family of small GTPases that controls intracellular vesicular trafficking in a compartment-specific manner. Mutations in the RAB39B gene cause intellectual disability comorbid with autism spectrum disorder and epilepsy, but the impact of RAB39B loss of function on synap...

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Detaylı Bibliyografya
Yayımlandı:Nat Commun
Asıl Yazarlar: Mignogna, Maria Lidia, Giannandrea, Maila, Gurgone, Antonia, Fanelli, Francesca, Raimondi, Francesco, Mapelli, Lisa, Bassani, Silvia, Fang, Huaqiang, Van Anken, Eelco, Alessio, Massimo, Passafaro, Maria, Gatti, Silvia, Esteban, José A., Huganir, Richard, D’Adamo, Patrizia
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Nature Pub. Group 2015
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4383008/
https://ncbi.nlm.nih.gov/pubmed/25784538
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms7504
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