The intellectual disability protein RAB39B selectively regulates GluA2 trafficking to determine synaptic AMPAR composition

RAB39B is a member of the RAB family of small GTPases that controls intracellular vesicular trafficking in a compartment-specific manner. Mutations in the RAB39B gene cause intellectual disability comorbid with autism spectrum disorder and epilepsy, but the impact of RAB39B loss of function on synap...

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Udgivet i:Nat Commun
Main Authors: Mignogna, Maria Lidia, Giannandrea, Maila, Gurgone, Antonia, Fanelli, Francesca, Raimondi, Francesco, Mapelli, Lisa, Bassani, Silvia, Fang, Huaqiang, Van Anken, Eelco, Alessio, Massimo, Passafaro, Maria, Gatti, Silvia, Esteban, José A., Huganir, Richard, D’Adamo, Patrizia
Format: Artigo
Sprog:Inglês
Udgivet: Nature Pub. Group 2015
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Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4383008/
https://ncbi.nlm.nih.gov/pubmed/25784538
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms7504
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