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Sam68 is cleaved by caspases under apoptotic cell death induced by ionizing radiation
The RNA-binding protein Sam68, a mitotic substrate of tyrosine kinases, has been reported to participate in the cell cycle, apoptosis, and signaling. In particular, overexpression of Sam68 protein is known to suppress cell growth and cell cycle progression in NIH3T3 cells. Although Sam68 is involved...
Tallennettuna:
| Julkaisussa: | J Radiat Res |
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| Päätekijät: | , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Oxford University Press
2015
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4380058/ https://ncbi.nlm.nih.gov/pubmed/25666188 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/jrr/rru113 |
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