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Sam68 is cleaved by caspases under apoptotic cell death induced by ionizing radiation

The RNA-binding protein Sam68, a mitotic substrate of tyrosine kinases, has been reported to participate in the cell cycle, apoptosis, and signaling. In particular, overexpression of Sam68 protein is known to suppress cell growth and cell cycle progression in NIH3T3 cells. Although Sam68 is involved...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:J Radiat Res
मुख्य लेखकों: Cho, Seong-Jun, Choi, Moo Hyun, Nam, Seon Young, Kim, Ji Young, Kim, Cha Soon, Pyo, Suhkneung, Yang, Kwang Hee
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: Oxford University Press 2015
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC4380058/
https://ncbi.nlm.nih.gov/pubmed/25666188
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/jrr/rru113
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