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Sam68 is cleaved by caspases under apoptotic cell death induced by ionizing radiation
The RNA-binding protein Sam68, a mitotic substrate of tyrosine kinases, has been reported to participate in the cell cycle, apoptosis, and signaling. In particular, overexpression of Sam68 protein is known to suppress cell growth and cell cycle progression in NIH3T3 cells. Although Sam68 is involved...
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| Publicado no: | J Radiat Res |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4380058/ https://ncbi.nlm.nih.gov/pubmed/25666188 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/jrr/rru113 |
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