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A primary cell model of HIV-1 latency that uses activation through the T cell receptor and return to quiescence to establish latent infection
A mechanistic understanding of HIV-1 latency depends upon a model system that recapitulates the in vivo condition of latently infected, resting CD4(+) T lymphocytes. Latency appears to be established after activated CD4(+) T cells, the principal targets of HIV-1 infection, become productively infect...
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| Publicado no: | Nat Protoc |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4378543/ https://ncbi.nlm.nih.gov/pubmed/25375990 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nprot.2014.188 |
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