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Sulfide Oxidation by a Noncanonical Pathway in Red Blood Cells Generates Thiosulfate and Polysulfides
A cardioprotectant at low concentrations, H(2)S is a toxin at high concentrations and inhibits cytochrome c oxidase. A conundrum in H(2)S homeostasis is its fate in red blood cells (RBCs), which produce H(2)S but lack the canonical mitochondrial sulfide oxidation pathway for its clearance. The sheer...
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| 出版年: | J Biol Chem |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Biochemistry and Molecular Biology
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4375485/ https://ncbi.nlm.nih.gov/pubmed/25688092 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M115.639831 |
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