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Sulfide Oxidation by a Noncanonical Pathway in Red Blood Cells Generates Thiosulfate and Polysulfides
A cardioprotectant at low concentrations, H(2)S is a toxin at high concentrations and inhibits cytochrome c oxidase. A conundrum in H(2)S homeostasis is its fate in red blood cells (RBCs), which produce H(2)S but lack the canonical mitochondrial sulfide oxidation pathway for its clearance. The sheer...
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| Publicado no: | J Biol Chem |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Biochemistry and Molecular Biology
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4375485/ https://ncbi.nlm.nih.gov/pubmed/25688092 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M115.639831 |
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