טוען...
CD39 deficiency in murine liver allografts promotes inflammatory injury and immune-mediated rejection
Adenosine triphosphate (ATP), an essential metabolic energy source, is released following cell apoptosis or necrosis. It acts as a damage-associated molecule pattern to stimulate innate immune cells. The ectonucleotidase CD39 regulates immune activation by hydrolysis of extracellular ATP. We have sh...
שמור ב:
| הוצא לאור ב: | Transpl Immunol |
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| Main Authors: | , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2015
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4368493/ https://ncbi.nlm.nih.gov/pubmed/25661084 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.trim.2015.01.003 |
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