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The Defect in the Hunter Syndrome: Deficiency of Sulfoiduronate Sulfatase
Skin fibroblasts cultured from patients affected with the Hunter syndrome are deficient in the activity of a protein, named the “Hunter corrective factor,” that is required for degradation of dermatan and heparan sulfates. We now show that this factor, purified from human urine, removes about 2% of...
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| Gepubliceerd in: | Proc Natl Acad Sci U S A |
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| Hoofdauteurs: | , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
National Academy of Sciences
1973
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC433682/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/4269173/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.70.7.2134 |
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