Senescence marker protein-30/gluconolactonase deficiency exacerbates diabetic nephropathy through tubular injury in a mouse model of type 1 diabetes

Lignende værker

• Pancreatic insulin release in vitamin C-deficient senescence marker protein-30/gluconolactonase knockout mice
  af: Senmaru, Takafumi, et al.
  Udgivet: (2012)
• Lepr(db/db) Mice with Senescence Marker Protein-30 Knockout (Lepr(db/db)Smp30(Y/−)) Exhibit Increases in Small Dense-LDL and Severe Fatty Liver Despite Being Fed a Standard Diet
  af: Kondo, Yoshitaka, et al.
  Udgivet: (2013)
• Senescence marker protein 30 functions as gluconolactonase in l-ascorbic acid biosynthesis, and its knockout mice are prone to scurvy
  af: Kondo, Yoshitaka, et al.
  Udgivet: (2006)
• Visit-to-Visit Blood Pressure Variability Is a Novel Risk Factor for the Development and Progression of Diabetic Nephropathy in Patients With Type 2 Diabetes
  af: Okada, Hiroshi, et al.
  Udgivet: (2013)
• Metabolically Healthy Obesity and Risk of Incident CKD
  af: Hashimoto, Yoshitaka, et al.
  Udgivet: (2015)