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Vpr overcomes macrophage-specific restriction of HIV-1 Env expression and virion production
The HIV-1 accessory protein Vpr enhances infection of primary macrophages through unknown mechanisms. Recent studies demonstrated that Vpr interactions with the cellular DCAF1-DDB1-CUL4 E3 ubiquitin ligase complex limit activation of innate immunity and interferon (IFN) induction. We describe a rest...
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| Publicat a: | Cell Host Microbe |
|---|---|
| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2014
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4269377/ https://ncbi.nlm.nih.gov/pubmed/25464830 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.chom.2014.10.014 |
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