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Inhibiting TNF-α signaling does not attenuate induction of endotoxin tolerance
Tumor necrosis factor-alpha (TNF-α) is a central mediator of inflammatory responses elicited by Toll-like receptor agonists, such as the Gram-negative bacterial outer membrane antigen lipopolysaccharide (LPS). TNF-α is responsible for altering vascular permeability and activating infiltrating inflam...
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| Publicado no: | J Inflamm Res |
|---|---|
| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Dove Medical Press
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4259568/ https://ncbi.nlm.nih.gov/pubmed/25506235 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2147/JIR.S75037 |
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