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Signaling from DNA mispairs to mismatch-repair excision sites despite intervening blockades
Mismatch-repair (MMR) systems promote genomic stability by correction of DNA replication errors. Thus, MMR proteins—prokaryotic MutS and MutL homodimers or their MutSα and MutLα heterodimer homologs, plus accessory proteins—specifically couple mismatch recognition to nascent-DNA excision. In vivo ex...
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| Publicado no: | EMBO J |
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| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2004
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC424355/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15103323/ https://ncbi.nlm.nih.govhttps://doi.org/10.1038/sj.emboj.7600153 |
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