Signaling from DNA mispairs to mismatch-repair excision sites despite intervening blockades

Mismatch-repair (MMR) systems promote genomic stability by correction of DNA replication errors. Thus, MMR proteins—prokaryotic MutS and MutL homodimers or their MutSα and MutLα heterodimer homologs, plus accessory proteins—specifically couple mismatch recognition to nascent-DNA excision. In vivo ex...

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Bibliografiska uppgifter

I publikationen:	EMBO J
Huvudupphov:	Wang, Huixian, Hays, John B
Materialtyp:	Artigo
Språk:	Inglês
Utgiven:	Nature Publishing Group 2004
Ämnen:	Article
Länkar:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC424355/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15103323/ https://ncbi.nlm.nih.govhttps://doi.org/10.1038/sj.emboj.7600153
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