Compensatory Motor Neuron Response to Chromatolysis in the Murine hSOD1(G93A) Model of Amyotrophic Lateral Sclerosis

We investigated neuronal self-defense mechanisms in a murine model of amyotrophic lateral sclerosis (ALS), the transgenic hSOD1(G93A), during both the asymptomatic and symptomatic stages. This is an experimental model of endoplasmic reticulum (ER) stress with severe chromatolysis. As a compensatory...

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Dettagli Bibliografici
Autori principali: Riancho, Javier, Ruiz-Soto, Maria, Villagrá, Nuria T., Berciano, Jose, Berciano, Maria T., Lafarga, Miguel
Natura: Artigo
Lingua:Inglês
Pubblicazione: Frontiers Media S.A. 2014
Soggetti:
Neuroscience
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4206191/
https://ncbi.nlm.nih.gov/pubmed/25374511
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2014.00346
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