Compensatory Motor Neuron Response to Chromatolysis in the Murine hSOD1(G93A) Model of Amyotrophic Lateral Sclerosis

We investigated neuronal self-defense mechanisms in a murine model of amyotrophic lateral sclerosis (ALS), the transgenic hSOD1(G93A), during both the asymptomatic and symptomatic stages. This is an experimental model of endoplasmic reticulum (ER) stress with severe chromatolysis. As a compensatory...

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Detalhes bibliográficos
Main Authors: Riancho, Javier, Ruiz-Soto, Maria, Villagrá, Nuria T., Berciano, Jose, Berciano, Maria T., Lafarga, Miguel
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2014
Assuntos:
Neuroscience
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4206191/
https://ncbi.nlm.nih.gov/pubmed/25374511
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2014.00346
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