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DNA strand breaks and TDP-43 mislocation are absent in the murine hSOD1(G93A) model of amyotrophic lateral sclerosis in vivo and in vitro
Mutations in the human Cu/Zn superoxide dismutase type-1 (hSOD1) gene are common in familial amyotrophic lateral sclerosis (fALS). The pathophysiology has been linked to, e.g., organelle dysfunction, RNA metabolism and oxidative DNA damage conferred by SOD1 malfunction. However, apart from metabolic...
Shranjeno v:
| izdano v: | PLoS One |
|---|---|
| Main Authors: | , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Public Library of Science
2017
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5568271/ https://ncbi.nlm.nih.gov/pubmed/28832631 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0183684 |
| Oznake: |
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