Endoplasmic reticulum stress induced by tunicamycin and thapsigargin protects against transient ischemic brain injury: Involvement of PARK2-dependent mitophagy

Transient cerebral ischemia leads to endoplasmic reticulum (ER) stress. However, the contributions of ER stress to cerebral ischemia are not clear. To address this issue, the ER stress activators tunicamycin (TM) and thapsigargin (TG) were administered to transient middle cerebral artery occluded (t...

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Autores principales: Zhang, Xiangnan, Yuan, Yang, Jiang, Lei, Zhang, Jingying, Gao, Jieqiong, Shen, Zhe, Zheng, Yanrong, Deng, Tian, Yan, Haijing, Li, Wenlu, Hou, Wei-Wei, Lu, Jianxin, Shen, Yao, Dai, Haibing, Hu, Wei-Wei, Zhang, Zhuohua, Chen, Zhong
Formato: Artigo
Lenguaje:Inglês
Publicado: Landes Bioscience 2014
Materias:
Basic Research Paper
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4198364/
https://ncbi.nlm.nih.gov/pubmed/25126734
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/auto.32136
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