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Endoplasmic reticulum stress induced by tunicamycin and thapsigargin protects against transient ischemic brain injury: Involvement of PARK2-dependent mitophagy

Transient cerebral ischemia leads to endoplasmic reticulum (ER) stress. However, the contributions of ER stress to cerebral ischemia are not clear. To address this issue, the ER stress activators tunicamycin (TM) and thapsigargin (TG) were administered to transient middle cerebral artery occluded (t...

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Detalhes bibliográficos
Main Authors: Zhang, Xiangnan, Yuan, Yang, Jiang, Lei, Zhang, Jingying, Gao, Jieqiong, Shen, Zhe, Zheng, Yanrong, Deng, Tian, Yan, Haijing, Li, Wenlu, Hou, Wei-Wei, Lu, Jianxin, Shen, Yao, Dai, Haibing, Hu, Wei-Wei, Zhang, Zhuohua, Chen, Zhong
Formato: Artigo
Idioma:Inglês
Publicado em: Landes Bioscience 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4198364/
https://ncbi.nlm.nih.gov/pubmed/25126734
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/auto.32136
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