Endoplasmic reticulum stress induced by tunicamycin and thapsigargin protects against transient ischemic brain injury: Involvement of PARK2-dependent mitophagy

Transient cerebral ischemia leads to endoplasmic reticulum (ER) stress. However, the contributions of ER stress to cerebral ischemia are not clear. To address this issue, the ER stress activators tunicamycin (TM) and thapsigargin (TG) were administered to transient middle cerebral artery occluded (t...

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Bibliografische gegevens
Hoofdauteurs: Zhang, Xiangnan, Yuan, Yang, Jiang, Lei, Zhang, Jingying, Gao, Jieqiong, Shen, Zhe, Zheng, Yanrong, Deng, Tian, Yan, Haijing, Li, Wenlu, Hou, Wei-Wei, Lu, Jianxin, Shen, Yao, Dai, Haibing, Hu, Wei-Wei, Zhang, Zhuohua, Chen, Zhong
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Landes Bioscience 2014
Onderwerpen:
Basic Research Paper
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4198364/
https://ncbi.nlm.nih.gov/pubmed/25126734
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/auto.32136
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