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Tissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia reperfusion injury
The murine cell surface protein Crry is a key complement regulator with similar activities to human membrane cofactor protein (MCP) and decay-accelerating factor. MCP plays a critical role in preventing complement-mediated tissue injury and its mutation has been implicated in several human kidney di...
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| Hauptverfasser: | , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2014
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4182132/ https://ncbi.nlm.nih.gov/pubmed/24850152 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ki.2014.103 |
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