טוען...
High glucose dephosphorylates serine 46 and inhibits p53 apoptotic activity
BACKGROUND: In response to diverse genotoxic stimuli p53 is activated as transcription factor to exert its tumor-suppressor function. P53 activation requires protein stabilization, nuclear localization and posttranslational modifications in key residues that may influence p53 selection of target gen...
שמור ב:
| Main Authors: | , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
BioMed Central
2014
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4181716/ https://ncbi.nlm.nih.gov/pubmed/25260780 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13046-014-0079-4 |
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