Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte death

Recurrent or sustained inflammation plays a causal role in the development and progression of left ventricular hypertrophy (LVH) and its transition to failure. Interleukin (IL)-18 is a potent pro-hypertrophic inflammatory cytokine. We report that induction of pressure overload in the rabbit, by cons...

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Detalhes bibliográficos
Main Authors: Yoshida, Tadashi, Friehs, Ingeborg, Mummidi, Srinivas, del Nido, Pedro J., Addulnour-Nakhoul, Solange, Delafontaine, Patrice, Valente, Anthony J., Chandrasekar, Bysani
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4157969/
https://ncbi.nlm.nih.gov/pubmed/25108227
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2014.07.007
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