Enhanced Orai1 and STIM1 expression as well as store operated Ca(2+) entry in therapy resistant ovary carcinoma cells

Mechanisms underlying therapy resistance of tumor cells include protein kinase Akt. Putative Akt targets include store-operated Ca(2+)-entry (SOCE) accomplished by pore forming ion channel unit Orai1 and its regulator STIM1. We explored whether therapy resistant (A2780cis) differ from therapy sensit...

Description complète

Enregistré dans:
Détails bibliographiques
Auteurs principaux: Schmidt, Sebastian, Liu, Guoxing, Liu, Guilai, Yang, Wenting, Honisch, Sabina, Pantelakos, Stavros, Stournaras, Christos, Hönig, Arnd, Lang, Florian
Format: Artigo
Langue:Inglês
Publié: Impact Journals LLC 2014
Sujets:
Research Paper
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC4148100/
https://ncbi.nlm.nih.gov/pubmed/25015419
Tags: Ajouter un tag
Pas de tags, Soyez le premier à ajouter un tag!

Documents similaires