Enhanced Orai1 and STIM1 expression as well as store operated Ca(2+) entry in therapy resistant ovary carcinoma cells

Mechanisms underlying therapy resistance of tumor cells include protein kinase Akt. Putative Akt targets include store-operated Ca(2+)-entry (SOCE) accomplished by pore forming ion channel unit Orai1 and its regulator STIM1. We explored whether therapy resistant (A2780cis) differ from therapy sensit...

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Bibliografski detalji
Glavni autori: Schmidt, Sebastian, Liu, Guoxing, Liu, Guilai, Yang, Wenting, Honisch, Sabina, Pantelakos, Stavros, Stournaras, Christos, Hönig, Arnd, Lang, Florian
Format: Artigo
Jezik:Inglês
Izdano: Impact Journals LLC 2014
Teme:
Research Paper
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4148100/
https://ncbi.nlm.nih.gov/pubmed/25015419
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