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Is JAK2(V617F) finally off the hook?
In this issue of Blood, Lamrani et al use various mouse models to demonstrate that the disease phenotype, rather than the JAK2(V617F) mutation, is responsible for the increased risk of thrombosis and bleeding in mouse models of myeloproliferative neoplasms (MPNs).(1)
Bewaard in:
| Hoofdauteurs: | , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Society of Hematology
2014
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4133501/ https://ncbi.nlm.nih.gov/pubmed/25124782 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2014-07-583856 |
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