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Is JAK2(V617F) finally off the hook?

In this issue of Blood, Lamrani et al use various mouse models to demonstrate that the disease phenotype, rather than the JAK2(V617F) mutation, is responsible for the increased risk of thrombosis and bleeding in mouse models of myeloproliferative neoplasms (MPNs).(1)

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Detalhes bibliográficos
Main Authors: Hers, Ingeborg, Poole, Alastair W.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society of Hematology 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4133501/
https://ncbi.nlm.nih.gov/pubmed/25124782
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2014-07-583856
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