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Inhibition of CXCR4 in CML cells disrupts their interaction with the bone marrow microenvironment and sensitizes them to nilotinib: Potentiation of nilotinib by CXCR4 antagonist

Drug resistance is a growing area of concern. It has been shown that a small, residual pool of leukemic CD34+ progenitor cells can survive in the marrow microenvironment of chronic myeloid leukemia (CML) patients after years of kinase inhibitor treatment. Bone marrow stroma has been implicated in th...

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Detalhes bibliográficos
Main Authors: Weisberg, Ellen, Azab, Abdel Kareem, Manley, Paul W., Kung, Andrew L., Christie, Amanda L., Bronson, Rod, Ghobrial, Irene M., Griffin, James D.
Formato: Artigo
Idioma:Inglês
Publicado em: 2011
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4124489/
https://ncbi.nlm.nih.gov/pubmed/22182920
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/leu.2011.360
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