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Potentiation of Nilotinib-mediated cell death in the context of the bone marrow microenvironment requires a promiscuous JAK inhibitor in CML

In this study, we show that conditioned media (CM) generated from bone marrow (BM)-derived mesenchymal stromal cells lead to BCR-ABL independent STAT3 activation. Activation of STAT3 is important not only for survival of CML cells but also for its protection against Nilotinib (NI), within the BM mic...

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Autors principals: Nair, Rajesh R., Tolentino, Joel H., Argilagos, Raul F., Zhang, Ling, Pinilla-Ibarz, Javier, Hazlehurst, Lori A.
Format: Artigo
Idioma:Inglês
Publicat: 2011
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3331955/
https://ncbi.nlm.nih.gov/pubmed/22209738
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.leukres.2011.12.002
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